Research into the causes of Atherosclerosis, the primary cause of heart disease, has been ongoing for many years. In the past 20 years or more, new light has been shed on not only the cause of this epidemic killer, but effective, natural methods of preventing and even reversing Atherosclerosis have been discovered. Some of the hundreds of studies on file have this to say about nutrients and heart disease:
….hyperhomocysteinemia is now generally accepted as an independent risk factor for coronary artery disease…. Its treatment with low doses of vitamins has proven to be both safe and effective. Archives of Internal Medicine 1997:157
“Dietary intakes of polyunsaturated fatty acids from seafood is associated with a reduced risk of primary cardiac arrest.” JAMA 1995 : 274“
These results suggest that intake of folate and vitamin B6, above the recommended dietary allowance may be important in the primary prevention of CHD among women. JAMA 1998: 279
“…it has been shown that coenzyme Q10 supplementation can be of value maintaining the integrity of the heart under the adverse cellular conditions. It would appear that the protection of the heard provided by CoQ10 may be related to its antioxidant capabilities although other factors cannot be ruled out…” Biological Trace Element Research 1993: 37
A selection of some of the hundred of studies used in this formulation follow:
1. McGill, HC. The geographic pathology of Atherosclerosis. Laboratory Investigation, Vol 18; 5.
2. The Pathogenesis of Atherosclerosis. Edited by RW Wissler and JC Geer. The Williams & Wilkins company, 1972.
3. Benditt, EP and Benditt, JM. Evidence for a monoclonal origin of human atherosclerotic plaques. Proceedings of the National Academy of Sciences. 70: (6) 1753-56, 1993.
4. Benditt, EP, Implications of the monoclonal character of human atherosclerotic plaques. Beitrage zur Pathologies 158: (4) 405-416.
5. Kyudkin, J. Dietary fat and dietary sugar in relation to ischemic heart disease and diabetes. Lancet .
6. Yudkin J. Levels of dietary sucrose in patients with occlusive atherosclerotic disease. Lancet, 6.8
7. Yudkin, J. Diet and coronary thrombosis. Lancet, 155-62. 1957. 8. Blankenhorn, DH., et al. The influence of diet on the appearance of new lesions in
human coronary arteries. JAMA 1990; 263: 1646-52.
9. Witztum, JL, The oxidation hypothesis of Atherosclerosis. Lancet, 344: 793-5. 1994.
10. Scharts, CJ., et al. The pathogenesis of Atherosclerosis: an overview. Clin Cariol. 14 (1): 1-16.
11. Ross, R. The pathogenesis of Atherosclerosis – An update. JEJM 314 (8): 488- 500.
12. Steinberg, D., et al. Beyond Cholesterol NEJM 320 (14): 915-24.
13. Goodnight, SH, et al. Polyunsaturated fatty acids and hyperlipidemia and thrombosis. Artiosclerosis, 2: 87-113.
14. Ginsberg H., et al. Induction of hypertriglyceridemia by a low fat diet. J Clin Endocrinol Metab. 42: 729-35.
15. Leaf, DA, Omega-3 fatty acids and coronary artery disease. Postrad Med 85(8) 237-42.
16. Rucknerova, O and Tulacek, J. Chelates in the treatment of occlusive arteriosclerosis. Vnitr Lek 18: 729-736.
17. Clarke, NE. Atherosclerosis, occlusive vascular disease and EDTA. Am J Cardiol 6:233-236.
18. Clarke NE. Treatment of occlusive vascular disease with disodium ethylene diamine tetraacetic acid (EDTA) Am J Med Sci 239: 732-744.
19. Wilder, LW., et al. Mobilization of atherosclerotic plaque calcium with EDTA utilizing the isolation-perfusion principle. Surger 52: 793-795.